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One study using nimodipine in a double-blind, coumadin metabolism placebo-controlled, crossover study in children showed no significant difference between the group receiving the active drug and the group receiving placebo.

Results are presented as mean ± SD of three independent experiments.

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Another explanation might be the presence of cytoplasmic stressors such as increased reactive oxygen species (ROS) that can induce DNA damage and mitochondrial response independent of Bcl-2 inhibition [25], [38]?

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